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顱內血腫英文ppt課件(編輯修改稿)

2026-01-31 23:36 本頁面

　

【文章內容簡介】 , cerebral edema may threaten life. ? Mannitol (1)g/kg body weight iv drips(fast) ? furosamide (Lasix) 40mg iv ? Glycerol 250 gtt (slow). ? Human albumin can also be used to increase osmotic pressure of colloid. Management of blood pressure ? It is of great importance to maintain blood pressure. If the blood pressure is not higher than 180/100 mm Hg, antihypertensive drugs is not necessary to use. Surgical operation ? Removal of the hematoma by evacuation or aspiration. Hematoma in cerebellum or near the cortical surface. Hemostasis ? Hemostatic therapy is not remended except for other hemorrhagic disorders. Cerebral protectives ? GABA, Nicholine(late stage), etc. Prevention and treatment of plications ? Gastric erosion, gastrointestinal hemorrhage, pneumonia TREATMENT stage (1)Rehabilitation (2)Control of hypertension (3)Prevention of recurrence PROGNOSIS ? The immediate prognosis is grave, some 70 to 75 percent of patients die in 1 to 30 days. PREVENTION of Hypertension of inducement Q: Which site of brain hemorrhage has not paralysis A. Basal ganglia B. Lobes (temporal and other lobes) C. Cerebellar hemisphere D. Pons E. Ventricles 02 SUBARACHNOID HEMORRHAGE ? DEFINITION OF TERM Spontaneous bleeding in the subarachnoid space. SUBARACHNOID HEMORRHAGE ? Traumatic: ? Spontaneous: SecondaryBlood in subarachnoid space is from the hematoma in brain tissue. PrimaryBlood in subarachnoid space originates from ruptured blood vessels in subarachnoid space(on surface of brain). ETIOLOGY Causes of subarachnoid hemorrhage: aneurysm or berry aneurysm malformation (AVM) ETIOLOGY arteriosclerosis: Hyalinosis, fibrinoid necrosis and formation of microaneurysms of arterioles and small arteries on brain surface. ETIOLOGY : Arteritis, Moyamoya disease, Hemorrhagic disorders, etc. cause (normal blood pressure, no aneurysm or AVM and no hemorrhagic and other disorders above). PATHOGENESIS AND PATHOLOGY 1. Ruptured blood vesselsbleeding in subarachnoid spacebrain edemaIntracranial hypertensionHernia of brainIschemia and necrosis of brain stemDeath 2. Active materials in blood vascular spasmcerebral ischemia and infarction CLINICAL PICTURE 1. Generally, mon occurrence age is 4070 years (lower than in cereb

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